Models by evolutionary biologists are not models
How Networks Are Revolutionizing Scientific (and Maybe Human) Thought By John Edward Terrell, Termeh Shafie and Mark Golitko | December 12, 2014 | Excerpt: “…evolutionary biologists […]
chemical
Pulses of olfactory/pheromonal input
Signal and noise: Spike correlations in the olfactory system by John Hewitt Excerpt: “The olfactory system is not so much a hard-wired telephone network where […]
Random mutations: nothing adaptive in 3 billion years
This is an open access article. Any advocate of adaptive evolution via random mutations can attempt to find evidence that I could not find by scanning it.
A "new" view of evolution sans mutations
Is there a simpler “proof” that makes those who think that random mutations cause adaptive evolution appear even more ridiculous?
Epigenetics and metabolism converge with pheromone production
Shall we attribute to him the inability to see that metabolism and epigenetics converge with food odors and the metabolism of nutrient chemicals to pheromones that act on precisely the same molecular mechanisms of adaptive evolution found in species from microbes to man?
Adaptive evolution: Genetic selection against some disorders
a model for adaptive evolution, for example, that is not nutrient chemical-dependent and pheromone-controlled?
A mathematical model of phenotypic cause, effects on genes, and affects on behavior
the only thing wrong with mathematical models of cause and effect is that they attribute indirect genetic effects, direct genetic effects, and affects on behavior to something unknown
A key mutational event? That's not very likely.
theorists argue that the 4.5 million interactive DNA switches in the human genome evolved via random mutations.
Genetic Influences on Epigenetically Effected Disease
By the time you get to 4 or 5 million nutrient-chemical and pheromone-controlled DNA switches in the human genome
Disorders of development: altered microRNA / messenger RNA balance
This model of systems biology (“biological embedding”) represents: Nutrient chemical-dependent and social stress-induced intracellular changes in microRNA and messenger RNA homeostasis