Random mutations and eyes: a pseudoquestion?

A participant on the ISHE’s yahoo group took a new “out-of-context” approach to the question: “…how [could] random mutations…cause development of eyes…”? (By out-of-context I mean that he uses quotes but does not indicate where the question in the quotes came from, then answers the question as if the context were unimportant.)

On 3/11/13 Sizemore wrote in msg #44875: “…random mutations may be related to phenotypic changes that can then be acted upon by the selective environment. Get it?

My response: No, I don’t “get it.” Is there a model for that? How are the random mutations related to phenotypic changes?

On 3/11/13 Sizemore wrote in msg #44875: “Another way that the genotype and phenotype may change is if the environment changes and begins to exert selection upon existing traits that are tied to genotype.

My response: In my model of cause and effect, that’s how nutrients epigenetically effect the genotype and how the metabolism of the nutrients to species-specific pheromones enables selection for existing and for novel traits that are tied to genotype.

I think the biological facts have become so obvious that even the most adamant proponents of random mutations theory are being forced to change to an accurate representation of cause and effect.

For example, watch how the moderator of the ISHE group changed his misrepresentations of the role of random mutations from 11/1/12 to 3/13/12.

On 11/1/12 Feierman wrote “Random mutations are the substrates upon which directional natural selection acts.

On 2/16/13 Feierman wrote: “Random gene mutation is the variance generator upon which natural selection operates.

On 2/23/13 Feierman wrote in the topic thread with the title Newman 2013 Pigeon Study Contradicts Darwinian Natural Selection: “…random genetic mutations generate the substrate upon which natural selection can act. Random genetic mutations create structural variations in protein enzymes…

On 3/11/13 in msg #44876 Feierman wrote:
“…random mutation, although the usual substrate for natural selection, is not a sufficient cause of natural selection. Random mutations are a contributing cause. They are not even an essential cause…

In my model of nutrient-dependent pheromone-controlled thermodynamics and thermoregulation of adaptive evolution, natural selection is for nutrients. The nutrients metabolize to pheromones that control reproduction. Receptor-mediated natural selection for nutrients enables adaptive evolution via pheromone-controlled receptor-mediated ecological, social, neurogenic, and socio-cognitive niche construction.

Now that others, like Feierman, realize random mutations “…are not even an essential cause…” discussion might being about how “…other mechanisms, like immigration or genetic drift, can increase the frequency of new alleles (genes) in a population.”

In my model, new alleles (genes) in a population are nutrient-dependent and pheromone-controlled in species from microbes to man.

See for example: Nutrient-dependent / Pheromone-controlled Adaptive Evolution

and Nutrient-dependent / Pheromone-controlled thermodynamics and thermoregulation

 

 

About James V. Kohl 1307 Articles
James Vaughn Kohl was the first to accurately conceptualize human pheromones, and began presenting his findings to the scientific community in 1992. He continues to present to, and publish for, diverse scientific and lay audiences, while constantly monitoring the scientific presses for new information that is relevant to the development of his initial and ongoing conceptualization of human pheromones. Recently, Kohl integrated scientific evidence that pinpoints the evolved neurophysiological mechanism that links olfactory/pheromonal input to genes in hormone-secreting cells of tissue in a specific area of the brain that is primarily involved in the sensory integration of olfactory and visual input, and in the development of human sexual preferences. His award-winning 2007 article/book chapter on multisensory integration: The Mind’s Eyes: Human pheromones, neuroscience, and male sexual preferences followed an award winning 2001 publication: Human pheromones: integrating neuroendocrinology and ethology, which was coauthored by disinguished researchers from Vienna. Rarely do researchers win awards in multiple disciplines, but Kohl’s 2001 award was for neuroscience, and his 2007 “Reiss Theory” award was for social science. Kohl has worked as a medical laboratory scientist since 1974, and he has devoted more than twenty-five years to researching the relationship between the sense of smell and the development of human sexual preferences. Unlike many researchers who work with non-human subjects, medical laboratory scientists use the latest technology from many scientific disciplines to perform a variety of specialized diagnostic medical testing on people. James V. Kohl is certified with: * American Society for Clinical Pathology * American Medical Technologists James V. Kohl is a member of: * Society for Neuroscience * Society for Behavioral Neuroendocrinology * Association for Chemoreception Sciences * Society for the Scientific Study of Sexuality * International Society for Human Ethology * American Society for Clinical Laboratory Science * Mensa, the international high IQ society