Two articles appeared on the same day in the psychiatry research yahoo group cue that link the sense of smell to atypical development via the ectoderm and neuronal “wiring” of the brain and atypical behavior in autism.
Food odor-associated atypical behavior: 1) “What happens when a child with autism refuses most foods?.” November 8th, 2013.
Pheromone-associated atypical behavior 2) “Social symptoms in autistic children may be caused by hyper-connected neurons.” November 7th, 2013.
Does anyone think in terms of systems biology instead of focusing on brain imaging and behaviors attributed to hormones downstream from THE biological core of behavior: gonadotropin releasing hormone (GnRH)?
A free “look inside” search for “Graziadei” in my 1995/2002 book (co-authored by the late Dr. Robert T. Francoeur) leads to the connection between GnRH, the olfactory bulbs, and brain development that continues to be ignored at a time when some people have obviously learned about the brain’s ectodermal origins.
Excerpted from an article posted to the cognitive neuroscience yahoo group on the same day: “Neuroscientists talk a lot about the functions of the 302, or more, neurons found in C. elegans. This worm also has some 50 glial cells, which like neurons, come from ectodermal precursor tissues.”
Shouldn’t others who are interested in psychiatry research and/or cognitive neuroscience begin to coordinate their efforts to understand disordered neuronal wiring via animal models of ecological, social, neurogenic, and socio-cognitive niche construction? The connections are as easy to make as counting from 1 to 3: Connections: Olfactory/pheromonal input links epigenetics to adaptations via 1) the ectoderm to 2) neuronal wiring and to 3) atypical behaviors in autism. Is the problem that you must start with olfactory/pheromonal input to make the connections to adaptations and to disorders?