By Carl Engelking | June 5, 2014 3:41 pm
Excerpt: “Together, the series of studies bolsters a branch of research called nutrigenetics, which focuses on understanding the way our genes affect our choice of foods and our body’s ability to process these foods.”
My comment (Discover did not approve it):
Nutrigenetics is nutritional epigenetics because the nutrients epigenetically effect cell type differentiation, which is how they change behaviors.
Nutrient stress and social stress induce receptor-mediated changes in behaviors associated with ecological variations that must result in ecological adaptations via cell type differentiation for individuals of different species to survive. The result is biodiversity.
What could possibly go wrong?
Olfactory receptors: Mosaic Epigenetic Dysregulation of Ectodermal Cells in Autism Spectrum Disorder
Are ASDs the disorders of nutrient-dependent cell type differentiation most closely linked to the epigenetic effects of pheromones on social behavior?
News sources often limit accurate representations of biologically based cause and effect and tout the pseudoscientific nonsense of population genetics. The Discover blog D-brief reported that genes are responsible for the development of our food preferences. My comment cited works that link experience-dependent de novo creation of taste receptors and olfactory receptors to the hormone-organized and hormone-activated development of food preferences across species from insects to humans.
If my comment is not published, uninformed readers will continue to assume that food preferences are genetically determined despite experimental evidence that clarifies what is currently known about nutritional epigenetics, which they call “nutrigenetics” because they do not understand how the epigenetic landscape becomes the physical landscape of DNA in the organized genomes of species from microbes to man.