Has anyone else detailed the required bottom-up/top-down reciprocity of the adaptively evolved gene, cell, tissue, organ, organ system pathway, which is exemplified in the honeybee model organism I used to link the adaptive evolution of microbes to man?
Isn’t the diet-responsive and presumably exercise- and pheromone-responsive hypothalamic neurogenic niche most likely to be involved in the cause and effect regulation of HDL via nutrient chemical-dependent epigenetic effects on gonadotropin releasing hormone?
“Do these people really think its a protein in semen that acts on the female brain? If so, is there a model for that
what if the epigenetic effects of pheromones on hormones associated with the physical trauma caused PTSD in women that affected subsequent behavior?
…you can now compare the science that’s behind the claims, unless the claims others make are for undisclosed active ingredients that somehow cause this affect on behavior (e.g., increased affection).
…chemical signals akin to species-specific pheromones are responsible for the ligand-receptor binding that enables the progression to colony formation as a more effective means of nutrient acquisition.
I will not dismiss the potential importance of oxytocin to effective treatment despite the fact that the mechanisms of its effectiveness remain unknown.
The epigenetic effects are on interactions among all the other neuronal systems and hormone secretion that interact with the GnRH neuronal system, which is central to all developmental differences in the brain of males and females.
Nutritional and social stress act via the same central neuronal system in mammals,
No mammal survives without the epigenetic effect of pheromones on species-specific differences in behavior, including sex differences in behavior.